参考文献
-
1) CEA-induced PI3K/AKT pathway activation through the binding of CEA to KRT1 contributes to oxaliplatin resistance in gastric cancer.
-
2) Brain Targeting Bacterial Extracellular Vesicles Enhance Ischemic Stroke Therapy via Efficient ROS Elimination and Suppression of Immune Infiltration.
-
3) Low miR-224-5p in exosomes confers colorectal cancer 5-FU resistance by upregulating S100A4.
-
4) Oligodendroglial precursor cells modulate immune response and early demyelination in a murine model of multiple sclerosis.
-
5) Deep Learning Enhanced Near Infrared-II Imaging and Image-Guided Small Interfering Ribonucleic Acid Therapy of Ischemic Stroke.
-
6) Nanosized Shikonin Disrupts Tumor-cell Mismatch Repair and Synergizes with Manganese to Sensitize Squamous Carcinoma to Immunotherapy.
-
7) A Noninvasive Nanoeyedrop Therapy for the Inhibition of Uveal Melanoma: Tetrahedral Framework Nucleic Acid-Based Bioswitchable MicroRNA Delivery System.
-
8) Antibacterial Peptides-Produced Cell Hydrogel Eliminates Intracellular Pathogens and Remodels Immune Microenvironment for Osteomyelitis Therapy.
-
9) Cascade-Type Microglial Pyroptosis Inhibitors for Enhanced Treatment of Cerebral Ischemia-Reperfusion Injury.
-
10) Multifunctional Nanomedicine for Targeted Atherosclerosis Therapy: Activating Plaque Clearance Cascade and Suppressing Inflammation.
-
11) UCP2 inhibition eliminates pancreatic β cell autoinflammation in type 2 diabetes mellitus with Islet-mitochondrial sequential targeting nanomedicines..
-
12) Phenazine biosynthesis-like domain containing protein (PBLD) and Cedrelone promote antiviral immune response by activating NF-ĸB.
製品: 1Year: 2025ジャーナル: NAT COMMUNImpactFactor: 15.700DOI: https://www.nature.com/articles/s41467-024-54882-y -
13) An eNOS-like nanomaterial for specific reversal of cerebral ischemia-reperfusion injury.
-
14) Injectable and Sprayable Thermoresponsive Hydrogel with Fouling-Resistance as an Effective Barrier to Prevent Postoperative Cardiac Adhesions.
-
15) C1q+ Macrophage-Tumor Cell Interaction Promoted Tumorigenesis via GPR17/PI3K/AKT Pathway Induced DNA Hypermethylation in Nasopharyngeal Carcinoma.